{"created":"2023-06-23T12:14:03.470042+00:00","id":2367,"links":{},"metadata":{"_buckets":{"deposit":"857b52c8-4615-4cab-aa55-ae3e59f5781a"},"_deposit":{"created_by":19,"id":"2367","owners":[19],"pid":{"revision_id":0,"type":"depid","value":"2367"},"status":"published"},"_oai":{"id":"oai:asahi-u.repo.nii.ac.jp:00002367","sets":["64:123"]},"author_link":["2826"],"item_10_creator_30":{"attribute_name":"教員氏名","attribute_type":"creator","attribute_value_mlt":[{"creatorNames":[{"creatorName":"竹村, 元三"}],"nameIdentifiers":[{"nameIdentifier":"2826","nameIdentifierScheme":"WEKO"}]}]},"item_10_date_36":{"attribute_name":"発行又は発表の年月","attribute_value_mlt":[{"subitem_date_issued_datetime":"2014-04"}]},"item_10_text_41":{"attribute_name":"PubMed番号","attribute_value_mlt":[{"subitem_text_value":"24531087"}]},"item_10_textarea_29":{"attribute_name":"業績分類","attribute_value_mlt":[{"subitem_textarea_value":"学術雑誌論文"}]},"item_10_textarea_31":{"attribute_name":"発行、発表雑誌等、又は発表学会等の名称","attribute_value_mlt":[{"subitem_textarea_value":"Intern Med."}]},"item_10_textarea_32":{"attribute_name":"巻","attribute_value_mlt":[{"subitem_textarea_value":"53"}]},"item_10_textarea_33":{"attribute_name":"号","attribute_value_mlt":[{"subitem_textarea_value":"4"}]},"item_10_textarea_34":{"attribute_name":"掲載ページ","attribute_value_mlt":[{"subitem_textarea_value":"315-9"}]},"item_10_textarea_35":{"attribute_name":"単著、共著の別","attribute_value_mlt":[{"subitem_textarea_value":"共著"}]},"item_10_textarea_42":{"attribute_name":"概要","attribute_value_mlt":[{"subitem_textarea_value":"A 40-year-old man who was referred to our hospital due to dyspnea was found to have high output cardiac failure on Swan-Ganz catheterization. An endomyocardial biopsy revealed cardiomyocyte hypertrophy with a vacuolar structure consistent with mitochondrial disease (MD). The patient was discharged, then readmitted for high output cardiac failure with hypotension and hyperlactacidemia. Treatment with cardiopulmonary support and hemodiafiltration gradually improved his general condition, although it resulted in ischemic necrosis of the right leg. The hyperlactacidemia completely resolved after amputation, and the high output cardiac failure has not recurred for two years. High output cardiac failure is rare in MD patients and is related to myocardial abnormalities and hyperlactacidemia."}]},"item_access_right":{"attribute_name":"アクセス権","attribute_value_mlt":[{"subitem_access_right":"metadata only access","subitem_access_right_uri":"http://purl.org/coar/access_right/c_14cb"}]},"item_language":{"attribute_name":"言語","attribute_value_mlt":[{"subitem_language":"eng"}]},"item_resource_type":{"attribute_name":"資源タイプ","attribute_value_mlt":[{"resourcetype":"other","resourceuri":"http://purl.org/coar/resource_type/c_1843"}]},"item_title":"Refractory high output heart failure in a patient with primary mitochondrial respiratory chain disease.","item_titles":{"attribute_name":"タイトル","attribute_value_mlt":[{"subitem_title":"Refractory high output heart failure in a patient with primary mitochondrial respiratory chain disease."}]},"item_type_id":"10","owner":"19","path":["123"],"pubdate":{"attribute_name":"公開日","attribute_value":"2015-02-20"},"publish_date":"2015-02-20","publish_status":"0","recid":"2367","relation_version_is_last":true,"title":["Refractory high output heart failure in a patient with primary mitochondrial respiratory chain disease."],"weko_creator_id":"19","weko_shared_id":-1},"updated":"2023-06-23T14:55:06.779478+00:00"}